Clinical treatment of subarachnoid hemorrhage with lumbar puncture needle

The secondary brain injury after subarachnoid hemorrhage often manifests as the following: (1) Blood coagulation at the base of the skull or ventricle prevents the return of cerebrospinal fluid, acute obstructive hydrocephalus, and deposition of hemoglobin and hemosiderin on the arachnoid membrane Particles can also cause the reflux of cerebrospinal fluid to be blocked, and the occurrence of communicative hydrocephalus and ventricular dilatation; (2) Subarachnoid red blood cell collapse can cause chemical meningitis, and the increase of cerebrospinal fluid increases intracranial pressure; (3) The vasoactive substances and histamine released by the blood can stimulate blood vessels and meninges, causing vasospasm; (4) The blood can stimulate the opening of calcium ion channels, causing intracellular metabolism disorders, and severe cases can lead to apoptosis of nerve cells.

Cerebrospinal fluid therapy has the following effects: (1) It can eliminate free kinin, 5-HT and other inflammatory substances in bloody cerebrospinal fluid, reduce the content of collagen in cerebrospinal fluid, and reduce the stimulation of red blood cells to the subarachnoid space, and the damage of cerebral effusion The physical and chemical components gradually returned to normal; (2) After the intracranial cerebrospinal fluid was drained, the intracranial pressure was reduced and the pain relief was accelerated; (3) The dynamics of the cerebrospinal fluid was changed to promote the flow of cerebrospinal fluid, reduce the occurrence of hydrocephalus, and avoid the arachnoid membrane Of adhesion.

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